"Hemicrania continua should not be classified as a trigeminal
autonomic cephalagia."

Source.
Department of neurology, Brigham and women's hospital,
Harvard medical school, Boston MA, USA.

Abstract.
BACKGROUND: The pain of the so-called functional or primary
headache disorders, such as tension headaches, migraine,
or cluster headaches, can be associated with autonomic
symptons that are localized in nature. the localized
autonomic symptons proberly involve higher centers
autonomic regulation, for example the hypothalamus,
for which there is support from funcional magnetic
resonance imaging studies.

METHOD: Hermicrania continua,a continuous, unilateral,
side-locked headache,absolutely responsive to preventive
treatment with indomethacin, is contrasted with so-called
medication-overuse headache, in which the paradoxical
situation exists of tremendous suffering despite excessive
use of abortive medications.

CONCLUSION: In classification, clinical presention trumps
experimental testing. Not only is there no basis to classify
hemicrania continua in the category of the so-called
trigeminal autonomic cephalagia  also the existance of
this category lacks solid foundation.

Hemicrania continua should UNQUESTIONABLY be classified
as a trigeminal autonomic cephalagia.

Source.
Universidade federal do Rio De Janerio, Brazil.

Abstract.
Hemicrania continua (HC) is  a well-known primary headache
The present version of the international classification of
headache disorders lists (HC) in the "other primary headache
group". however, evidence has emerged demonstrating
that (HC)  is a phenotype that belongs to the trigeminal
autonomic cephalagias together with cluster headaches
paroxysmal hemicrania (PH), and short-lasting unilateral
neuralgiform headache attacks with conjunctival
injection and tearing.this is supported by a  common
general clinical picture paroxysmal unilateral, side-locked
headaches located to the ocular frontal, and/or temporal
regions, accompanied by ipsilateral autonomic disfunctions
including for example, tearing and conjunctional
injenction. Apart from the remarkable clinical similarities,
the absolute and incomparable effect of indomethacin in (HC) parallels the effect of this drug in (PH), suggesting
a shared core pathogenesis. finally, neuroimage findings
demostrate a posterior hypothalamic activation in (HC)
similary to cluster headaches. (PH), and short lasting,
unilateral neuralgiform headache attacks with conjunctival
injection and tearing. Taken together, data indicate that
(HC) is certainly a type of trigeminal autonomic cephalagia
that should no longer be placed in a group of miscellaneous
primary headache disorders.


Does this mean we are getting closer to an answer,
headaches that mimic cluster headaches, or is the
jury still out?

Thanks for that info.

Hoppy