CH and possible sleep apnea

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Posted by Thomas/Munich ( on March 01, 2001 at 14:45:33:

In Reply to: New here and in need of help posted by Stacey on February 27, 2001 at 12:03:39:

Hi Stacey,
I would like to stress that untill now nobody can
interprete (as far as I know...) the foundings of R.D.Chervin from the University of Michigan con-
cerning CH and sleep apnea, and I don't know whethwer you can influence CH by CPAP breathing or not - anyway, if you suffer from a sleep apnea
then you need a CPAP ventilator at home and then look and inform us if it helped against CH, too,
at least in your case.
Concerning Bob P.'s comment: I don't beleave anything, too, and I can't interprete a possible connection between p e r i o d i c a l CH and sleep apnea, but let's look...and I didn't say that e v e r y CH sufferer also suffers from sleep apnea, either.

BTW:Look at the abstract of R.D.Chervin below, published in the journal "Neurology" last year, you also can show it your treating doctor ->

---------------------------------------------------"NEUROLOGY" 2000;54:2302-2306 -----------------------------


Sleep disordered breathing in patients with cluster headache
R. D. Chervin, MD, MS, S. Nath Zallek, MD, X. Lin, PhD, J. M. Hall, MA, MS, N. Sharma, BS and K. M. Hedger, RN, BSN
From the Sleep Disorders Center, Department of Neurology (Drs. Chervin and Nath Zallek, J.M. Hall, N. Sharma, and K.M. Hedger) and the Department of Biostatistics (Dr. Lin), University of Michigan, Ann Arbor, MI.

Address correspondence and reprint requests to Dr. Ronald D. Chervin, Sleep Disorders Center, University Hospital 8D8702, Box 0117, 1500 E. Medical Center Dr., Ann Arbor, MI 48109-0117; e-mail:

OBJECTIVE: To study subjects with active or inactive cluster headache (CH) for occult sleep disordered breathing (SDB).

BACKGROUND: CH frequently occurs during sleep. The authors previously found that symptoms of SDB predicted reported occurrence of CH in the first half of the night, which suggested that CH could be triggered in some cases by unrecognized SDB.

METHODS: The authors performed polysomnography in 25 adults (22 men) with CH. Subjects were not selected for any sleep-related complaint. In addition to standard measures, studies included monitoring of end-tidal carbon dioxide (n = 22), and esophageal pressure (n = 20).

RESULTS: The rate of apneas and hypopneas per hour of sleep was >5 in 20 subjects (80%; 95% CI, 64% to 96%), minimum oxygen saturation was <90% in 10 subjects, maximum negative esophageal pressure ranged from -13 to -65 cm H2O, and maximum end-tidal carbon dioxide was 50 mm Hg in eight subjects. The eight subjects with active (versus inactive) CH at the time of study had higher maximum end-tidal carbon dioxide levels (50 ± 3 versus 44 ± 5 mm Hg; p = 0.0007). More severe oxygen desaturation was associated with reports that CH typically occurred in the first half of the nocturnal sleep period (p = 0.008).

CONCLUSIONS: SDB occurred in the majority of patients with CH. Evaluation of a patient with CH should include consideration that SDB may be present.

Key words: Cluster headache—Obstructive sleep apnea—Polysomnography—Hypercapnia—Hypoxemia.

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